Author

G. Filiz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

P. Daniel· 2 shared T. Mantamadiotis· 2 shared A. H. Kaye· 1 shared C. Pouton· 1 shared D. Brown· 1 shared D. Flanagan· 1 shared E. Vincan· 1 shared J. Haynes· 1 shared

Activity by Year

2017-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersPathology

P. Daniel

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2 papers

Recent·Most cited

Intratumoral MAPK and PI3K signaling pathway heterogeneity in glioblastoma tissuecorrelates with selective CREB signaling and specific functional gene signatures

P. Daniel, G. Filiz, M. Tymms, R. Ramsay, A. H. Kaye, S. Stylli, T. Mantamadiotis

The paper identifies intratumoral heterogeneity in glioblastoma by analyzing MAPK and PI3K pathway activation and their correlation with CREB signaling and gene signatures.

bioRxiv · Apr 20, 2018Pathology

PI3K Activation In Neural Stem Cells Drives Tumorigenesis Which Can Be Suppressed By Targeting CREB

P. Daniel, G. Filiz, D. Brown, M. Christie, P. Waring, Y. Zhang, J. Haynes, C. Pouton, D. Flanagan, E. Vincan, T. Johns, K. Montgomery, W. Phillips, T. Mantamadiotis

This study shows that PI3K pathway activation in neural stem cells initiates tumorigenesis, and targeting CREB reduces tumor growth in a mouse model of high-grade glioma.

bioRxiv · May 30, 2017Cancer Biology