Author

G. Krilis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Caporali· 1 shared A. M. Kilpatrick· 1 shared A. Pellicoro· 1 shared C. Bain· 1 shared C. Grenier· 1 shared D. E. W. Livingstone· 1 shared D. Severs· 1 shared E. J. Hoorn· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPathology

A. Caporali

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Author profile →

2 papers

Recent·Most cited

MC1R reduces scarring and rescues stalled healing in a preclinical chronic wound model

Y. Nawilaijaroen, H. R. Rocliffe, S. Austin-Williams, G. Krilis, A. Pellicoro, K. Zhou, Y. Ji, C. Bain, A. M. Kilpatrick, Y. Chen, M. Crichton, Z. Huang, S. J. Forbes, A. Caporali, J. L. Cash

This study shows that MC1R agonist reduces scarring and improves healing in a chronic wound model by enhancing angiogenesis, lymphangiogenesis, and reducing inflammation.

bioRxiv · Nov 30, 2022 · PreprintPathology

High salt intake activates the hypothalamic-pituitary-adrenal axis, amplifies the stress response, and alters tissue glucocorticoid exposure in mice

H. M. Costello, G. Krilis, C. Grenier, D. Severs, J. R. Ivy, M. Nixon, M. C. Holmes, D. E. W. Livingstone, E. J. Hoorn, N. Dhaun, M. A. Bailey

This study shows that high salt intake in mice activates the HPA axis, increases glucocorticoid exposure in multiple tissues, and enhances stress responses.

bioRxiv · Mar 4, 2022Physiology