Author

A. Caporali

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Assay:clinical cohort / trial· 1 Approach:deep learning· 1

Frequent Co-Authors

M. Meloni· 2 shared A. C. Thomas· 1 shared A. G. Rossi· 1 shared A. M. Kilpatrick· 1 shared A. Pellicoro· 1 shared A. Thomson· 1 shared A.-M. Spiroski· 1 shared C. Bain· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

M. Meloni

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MC1R reduces scarring and rescues stalled healing in a preclinical chronic wound model

Y. Nawilaijaroen, H. R. Rocliffe, S. Austin-Williams, G. Krilis, A. Pellicoro, K. Zhou, Y. Ji, C. Bain, A. M. Kilpatrick, Y. Chen, M. Crichton, Z. Huang, S. J. Forbes, A. Caporali, J. L. Cash

This study shows that MC1R agonist reduces scarring and improves healing in a chronic wound model by enhancing angiogenesis, lymphangiogenesis, and reducing inflammation.

bioRxiv · Nov 30, 2022 · PreprintPathology

Contractile reprogramming of cardiac pericytes by MEK inhibition promotes arteriologenesis of the ischemic heart

E. Avolio, R. Katare, A. C. Thomas, A. Caporali, D. Schwenke, M. Meloni, M. Caputo, P. Madeddu

This study shows that MEK inhibition reprograms cardiac pericytes into contractile cells, promoting arteriologenesis in ischemic hearts.

bioRxiv · Nov 10, 2020 · PreprintCell Biology

Eosinophil deficiency promotes aberrant repair and adverse remodelling following acute myocardial infarction

I. S. Toor, D. Ruckerl, I. Mair, R. Ainsworth, M. Meloni, A.-M. Spiroski, J. M. Felton, A. Thomson, A. Caporali, T. Keeble, K. Tang, A. G. Rossi, D. E. Newby, J. E. Allen, G. A. Gray

This study shows that eosinophil deficiency after myocardial infarction leads to adverse cardiac remodeling, which can be mitigated by IL-4 therapy.

bioRxiv · Sep 9, 2019Immunology