Author

Gaia Consonni

University of Milan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:protein sequence· 1

Frequent Co-Authors

Antonio GaleoneiD· 3 shared Thomas VaccariiD· 3 shared Hamed Jafar‐NejadiD· 2 shared Seung Yeop HaniD· 2 shared Aglaia VignoliiD· 1 shared Alessandro MineoiD· 1 shared Ashutosh N. PandeyiD· 1 shared Benjamin A. StoryiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersDevelopmental Biology

Antonio GaleoneORCID

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3 papers

Recent·Most cited

TUSC3 serves as a rate-limiting gatekeeper of a glycan-mediated ER Triage Checkpoint for BMP4/Dpp

Antonio Galeone, Emilio Solazzo, Francesco Lavezzari, Seung Yeop Han, Gaia Consonni, Bruna My, Riccardo Rizzo, Giuseppe Gigli, Hamed Jafar‐Nejad, Thomas Vaccari

The paper identifies TUSC3 as a gatekeeper regulating glycan trimming of BMP4/Dpp to control ERQC entry and BMP signaling.

Cell Reports · Jun 30, 2026Peer-reviewedDevelopmental Biology

Mesodermal-specific MECP2 expression in Drosophila induces visceral and skeletal muscle defects rescued by butyrate supplementation

Gaia Consonni, Mustafa Kaan Kozluca, Maria Cristina Gagliani, Alessandro Mineo, Katia Cortese, Irene Miguel‐Aliaga, Aglaia Vignoli, Elisa Borghi, Antonio Galeone, Thomas Vaccari

This study shows that mesodermal-specific MECP2 overexpression in Drosophila causes muscle and gut defects, which are rescued by butyrate and valproate.

bioRxiv · Dec 18, 2025 · PreprintGenetics

Gut barrier defects, increased intestinal innate immune response, and enhanced lipid catabolism drive lethality in N-glycanase 1 deficient Drosophila

Ashutosh N. Pandey, Antonio Galeone, Seung Yeop Han, Benjamin A. Story, Gaia Consonni, William F. Mueller, Lars Michael Steinmetz, Thomas Vaccari, Hamed Jafar‐Nejad

This study shows that loss of Drosophila N-glycanase 1 causes gut barrier defects, leading to immune activation and lipid catabolism, which contribute to lethality through non-infectious mechanisms.

bioRxiv · Apr 7, 2023 · PreprintDevelopmental Biology