Author

H. S. Kulkarni

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. E. Gelman· 3 shared L. Ma· 3 shared M. Cano· 3 shared D. Kreisel· 2 shared D. Scozzi· 2 shared D. Zhou· 2 shared J. A. O'Halloran· 2 shared J. McPhatter· 2 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersImmunology

A. E. Gelman

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Lung epithelial cell-derived C3 protects against pneumonia-induced lung injury

A. N. Ozanturk, S. K. Sahu, D. H. Kulkarni, L. Ma, R. A. Barve, J. McPhatter, L. Garnica, L. Dannull, X. Wu, S. L. Brody, J. P. Atkinson, H. S. Kulkarni

The paper shows lung-derived C3 reduces pneumonia-induced lung injury through the Factor B pathway, independent of circulating C3 levels.

bioRxiv · Feb 3, 2022Immunology

Reprogramming Alveolar Macrophage Responses to TGF-β Reveals CCR2+ Monocyte Activity that Promotes Bronchiolitis Obliterans Syndrome

Z. Liu, F. Liao, J. Zhu, D. Zhou, G. S. Heo, H. P. Luehmann, D. Scozzi, A. Parks, R. Hachem, D. Byers, L. K. Tague, H. S. Kulkarni, M. Cano, B. W. Wong, W. Li, , A. E. Gelman

This study identifies alveolar macrophage reprogramming by ECP that reduces TGF-β bioavailability and prevents BOS by inhibiting CCR2+ monocyte-driven inflammation.

bioRxiv · Jan 27, 2022 · PreprintImmunology

Increased complement activation is a distinctive feature of severe SARS-CoV-2 infection.

L. Ma, S. K. Sahu, M. Cano, V. Kuppuswamy, J. Bajwa, J. McPhatter, A. B. Pine, M. M. Meizlish, G. Goshua, C. H. Chang, H. Zhang, C. Price, P. Bahel, H. M. Rinder, T. Lei, , H. S. Kulkarni

The paper shows that complement activation is higher in severe SARS-CoV-2 infection compared to influenza and non-COVID respiratory failure, with the alternative pathway linked to worse outcomes.

bioRxiv · Feb 22, 2021Immunology

Circulating Mitochondrial DNA is an Early Indicator of Severe Illness and Mortality from COVID-19

D. Scozzi, M. Cano, L. MA, D. Zhou, J. H. Zhu, J. A. O'Halloran, C. Goss, A. Rauseo, Z. Liu, V. Peritore, M. Rocco, A. Ricci, R. Amodeo, L. Aimati, M. Ibrahim, , A. E. Gelman

This study shows that high levels of circulating mitochondrial DNA predict severe illness and mortality in COVID-19 patients.

bioRxiv · Jul 30, 2020Immunology