Author

H. S. Ryu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

B. S. Hong· 1 shared D. Kim· 1 shared D. Lee· 1 shared D.-Y. Noh· 1 shared G. Park· 1 shared H.-B. Lee· 1 shared H.-G. Moon· 1 shared H.-J. Sung· 1 shared

Activity by Year

2016-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

B. S. Hong

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2 papers

Recent·Most cited

Widespread multi-targeted therapy resistance via drug-induced secretome fucosylation

M. B. D. Aldonza, J. Cha, I. Yong, J. Ku, D. Lee, P. Sinitcyn, R.-E. Cho, R. D. Delos Reyes, D. Kim, H.-J. Sung, S. Kim, M. Kang, Y. Ku, G. Park, H. S. Ryu, , Y. Kim

This study identifies drug-induced secretome fucosylation as a mechanism of resistance to targeted therapies across multiple cancers.

bioRxiv · Apr 21, 2021 · PreprintCancer Biology

Transition into inflammatory cancer-associated adipocytes in breast cancer microenvironment requires microRNA regulatory mechanism

J. Lee, H. S. Ryu, B. S. Hong, H.-B. Lee, M. Lee, I. A. Park, J. Kim, W. Han, D.-Y. Noh, H.-G. Moon

The paper identifies de-differentiated, inflammatory cancer-associated adipocytes in breast cancer and shows that miR-5112 promotes IL6 expression through targeting Cpeb1.

bioRxiv · Nov 23, 2016Cancer Biology