Author

H.-J. Tsai

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

A. A. R. Tobian· 1 shared A. L. Cox· 1 shared A. L. Mammen· 1 shared A. N. Baer· 1 shared A. Wilbon· 1 shared A. Z. Rosenberg· 1 shared B. Keevan· 1 shared B. Parekkadan· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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1 shared paperImmunology

A. A. R. Tobian

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Quantifying the mechanical properties of yeast Candida albicans using atomic force microscopy-based force spectroscopy

C. R. Jones, Z. J. Zhang, H.-J. Tsai

The paper describes a label-free atomic force microscopy method to measure the physical properties of Candida albicans in response to host stress and antifungal drugs.

bioRxiv · Oct 7, 2022 · PreprintMicrobiology

Neutralizing IFNL3 Autoantibodies in Severe COVID-19 Identified Using Molecular Indexing of Proteins by Self-Assembly

J. J. Credle, J. Gunn, P. Sangkhapreecha, D. R. Monaco, X. A. Zheng, H.-J. Tsai, A. Wilbon, W. R. Morgenlander, Y. Dong, S. Jayaraman, L. Tosi, B. Parekkadan, A. N. Baer, M. Roederer, E. M. Bloch, , H. B. Larman

MIPSA identifies neutralizing IFNL3 autoantibodies in severe COVID-19 patients by profiling autoantibodies against DNA-barcoded proteins.

bioRxiv · Mar 2, 2021 · PreprintImmunology

Oestrogen promotes innate immune evasion of Candida albicans through inactivation of the alternative complement system

P. Kumwenda, F. Cottier, B. Keevan, H. Gallagher, H.-J. Tsai, R. A. Hall

This study shows oestrogen enhances Candida albicans virulence by inactivating the alternative complement system through Factor H acquisition.

bioRxiv · Jul 22, 2020 · PreprintMicrobiology