Author

Helia Samani

University of California, Berkeley

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 1

Frequent Co-Authors

Alex ZilinskasiD· 2 shared Carolina AgudeloiD· 2 shared Sarah A. StanleyiD· 2 shared Amir BalakhmetiD· 1 shared Andrea Anaya-SancheziD· 1 shared Daniel A. PortnoyiD· 1 shared Douglas FoxiD· 1 shared Heyuan Michael NiiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

Alex ZilinskasORCID

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2 papers

Recent·Most cited

Mycobacterium tuberculosis suppresses protective Th17 responses during infection

Alex Zilinskas, Amir Balakhmet, Douglas Fox, Heyuan Michael Ni, Carolina Agudelo, Helia Samani, Sarah A. Stanley

This study identifies factors by which Mycobacterium tuberculosis suppresses Th17 responses during infection, showing that ESX-1 and PDIM inhibit IL-23 production in dendritic cells.

eLife · Oct 23, 2026Peer-reviewedImmunology

Methylglyoxal is an antibacterial effector produced by macrophages during infection

Andrea Anaya-Sanchez, Samuel B. Berry, Scott Espich, Alex Zilinskas, Phuong M. Tran, Carolina Agudelo, Helia Samani, K. Heran Darwin, Daniel A. Portnoy, Sarah A. Stanley

This study shows that methylglyoxal, a byproduct of aerobic glycolysis in infected macrophages, acts as an antimicrobial effector that enhances host defense against bacterial pathogens.

Cell Host & Microbe · Jun 23, 2025Peer-reviewedMicrobiology