Author

Huayang Yao

NIHR Imperial Biomedical Research Centre

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Reed· 1 shared Alison E. JohniD· 1 shared Bin LiuiD· 1 shared Elizabeth Pyman· 1 shared Gisli R. JenkinsiD· 1 shared Greg ContentoiD· 1 shared Iain D. StewartiD· 1 shared Iyobel Kibreab· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenetics

A. Reed

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1 paper

Recent·Most cited

Targeting AKAP13 RhoGEF activity ameliorates pro-fibrotic phenotypes driven by the IPF associated AKAP13 risk variant

Bin Liu, James Michael May, Greg Contento, Sara Gangi, Louise A. Organ, Elizabeth Pyman, Iyobel Kibreab, Lan Zhao, Huayang Yao, Rachel Clare Chambers, Iain D. Stewart, A. Reed, Gisli R. Jenkins, Alison E. John

This study shows that an AKAP13 genetic variant promotes fibrosis by altering epithelial signaling, and inhibiting AKAP13's RhoGEF activity reduces fibrotic markers in IPF models.

bioRxiv · Jan 23, 2026 · PreprintGenetics