Author

Ilona Skatulla

University of Freiburg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1

Frequent Co-Authors

Miriam SchmidtsiD· 3 shared Sebastian J. ArnoldiD· 3 shared Anna Klawonn· 2 shared Chiara M. SchröderiD· 2 shared Oliver SchillingiD· 2 shared Stefan TholeniD· 2 shared Zeineb Bakey· 2 shared Anna KöttgeniD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersCell Biology

Miriam SchmidtsORCID

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Author profile →

3 papers

Recent·Most cited

Distinct effects of hypomorphic IFT and dynein-2 skeletal ciliopathy disease alleles on chondrogenic differentiation, ECM composition and wnt signalling in ATDC5 derived cartilage-like organoids

Anna Klawonn, Himanshu Himanshu, Zeineb Bakey, Stefan Tholen, Ilona Skatulla, Yong Li, Chiara M. Schröder, Sebastian J. Arnold, Anna Köttgen, Oliver Schilling, Miriam Schmidts

This study examines how hypomorphic IFT and dynein-2 mutations affect chondrogenic differentiation, ECM composition, and Wnt signaling in ATDC5 organoids, revealing genotype-dependent changes in signaling pathways and ECM.

bioRxiv · May 25, 2026 · PreprintCell Biology

Deep phenotyping of ATDC5-derived in vitro cartilage organoids

Anna Klawonn, Stefan Tholen, Ilona Skatulla, Chiara M. Schröder, Sebastian J. Arnold, Oliver Schilling, Miriam Schmidts

This study performs time-resolved transcriptomic and ECM proteomic analyses of differentiating ATDC5 cells to characterize chondrogenesis and cartilage ECM formation.

bioRxiv · Mar 18, 2026 · PreprintCell Biology

Base editing derived models of human WDR34 and WDR60 disease alleles replicate retrograde IFT and hedgehog signaling defects and suggest disturbed Golgi protein transport

Dinu Antony, Elif Yılmaz Güleç, Zeineb Bakey, Isabel Schüle, Gwang-Jin Kim, Ilona Skatulla, Han G. Brunner, Sebastian J. Arnold, Miriam Schmidts

bioRxiv · Mar 14, 2022 · PreprintGenetics