Author

Gwang-Jin Kim

University of FreiburgORCID 0000-0002-2753-0883Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:gene regulatory network inference· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Sebastian J. ArnoldiD· 3 shared Chiara M. SchröderiD· 2 shared Simone ProbstiD· 2 shared Alexandra E. WehmeyeriD· 1 shared Alexis HofherriD· 1 shared Dinu Antony· 1 shared Elif Yılmaz GüleçiD· 1 shared Han G. BrunneriD· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersDevelopmental Biology

Sebastian J. ArnoldORCID

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3 papers

Recent·Most cited

Developmental BiologyDevelopmental CellPeer-reviewedAug 25, 2023

Eomes restricts Brachyury functions at the onset of mammalian gastrulation

Katrin M. Schüle, Jelena Weckerle, Simone Probst…·Sebastian J. Arnold

The paper investigates the distinct gene-regulatory functions of Eomes and Brachyury in mouse embryos and stem cells, showing Eomes restricts Brachyury activity to ensure proper lineage specification.

39 citations

mousestatistical model

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GeneticsbioRxivPreprintMar 14, 2022

Base editing derived models of human WDR34 and WDR60 disease alleles replicate retrograde IFT and hedgehog signaling defects and suggest disturbed Golgi protein transport

Dinu Antony, Elif Yılmaz Güleç, Zeineb Bakey…·Miriam Schmidts

1 citation

humanbulk RNA-seq

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Developmental BiologyNature Cell BiologyPeer-reviewedDec 1, 2019

Eomes and Brachyury control pluripotency exit and germ layer segregation by changes of chromatin state

Jelena Tošić, Gwang-Jin Kim, Mihael Pavlovic…·Sebastian J. Arnold

This study shows that Eomes and Brachyury control pluripotency exit and germ layer segregation by altering chromatin states, with Tbx factors repressing pluripotency and NE gene programs while enabling ME differentiation.

121 citations

mouseChIP-seq

𝕏 PostBluesky