Author

J. A. Tuazon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

D. M. Jones· 2 shared K. A. Read· 2 shared K. J. Oestreich· 2 shared M. D. Powell· 2 shared R. T. Warren· 2 shared A. Ali· 1 shared A. G. Freud· 1 shared A. Saadey· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

D. M. Jones

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Author profile →

3 papers

Recent·Most cited

Eos promotes TH2 differentiation by propagating the IL-2/STAT5 signaling pathway.

J. A. Tuazon, K. A. Read, B. K. Sreekumar, M. J. Yaeger, S. Varikuti, D. M. Jones, R. T. Warren, M. D. Powell, M. N. Rasheed, E. G. Duncan, L. M. Childs, K. M. Gowdy, K. J. Oestreich

This study identifies Eos as a positive regulator of TH2 cell differentiation by enhancing IL-2/STAT5 signaling in CD4+ T cells.

bioRxiv · Nov 2, 2022 · PreprintImmunology

Aiolos modulates the TFH and CD4-CTL differentiation programs via reciprocal regulation of the Zfp831/TCF-1/Bcl-6 axis and CD25

K. A. Read, D. M. Jones, S. Pokhrel, E. D. S. Hales, A. Varkey, J. A. Tuazon, C. D. Eisele, O. Abdouni, A. Saadey, R. T. Warren, M. D. Powell, J. M. Boss, E. A. Hemann, J. S. Yount, H. E. Ghoneim, , K. J. Oestreich

This study identifies Aiolos as a regulator of TFH and CD4-CTL differentiation, showing it modulates the Zfp831/TCF-1/Bcl-6 axis and suppresses the cytotoxic gene program.

bioRxiv · May 18, 2022 · PreprintImmunology

Natural killer cell immunosuppressive function requires CXCR3-dependent redistribution within lymphoid tissues

A. Ali, L. M. Canaday, H. A. Feldman, H. Cevik, M. T. Moran, S. Rajaram, N. Lakes, J. A. Tuazon, H. Seelamneni, D. Kirshnamurthy, E. Blass, D. H. Barouch, S. N. Waggoner

The paper shows that CXCR3 and type I IFN are critical for NK cell redistribution to T-cell zones, enabling suppression of antiviral T cells during infection.

bioRxiv · May 11, 2021Immunology