Author

J. B. Williams

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. C. Anderson· 1 shared A. Droit· 1 shared A. L. Kley· 1 shared A. P. Yeola· 1 shared A.-C. Villani· 1 shared C. Joly-Beauparlant· 1 shared C. S. Moore· 1 shared C. Stingley· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. C. Anderson

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Resident memory T cell development is associated with AP-1 transcription factor upregulation across anatomical niches.

N. P. Smith, Y. Yan, Y. Pan, J. B. Williams, K. Manakongtreecheep, S. Pant, J. Zhao, T. Tian, T. Pan, C. Stingley, K. Wu, J. Zhang, A. L. Kley, P. K. Sorger, A.-C. Villani, T. S. Kupper

This study identifies AP-1 transcription factors as key regulators of TRM development across tissues using scRNA-seq and ATAC-seq, revealing a novel mechanism for TRM reactivation.

bioRxiv · Sep 29, 2023 · PreprintImmunology

Male sex chromosomal complement exacerbates the pathogenicity of Th17 cells in a chronic model of CNS autoimmunity.

P. M. I. A. Doss, J. Baillargeon, A. P. Yeola, J. B. Williams, M. Leclercq, C. Joly-Beauparlant, M. Alpaugh, A. C. Anderson, A. Droit, H. Lassmann, C. S. Moore, M. Rangachari

This study shows male sex chromosomal complement increases Th17 cell pathogenicity in CNS autoimmunity, independent of hormones, through downregulation of Jarid1c.

bioRxiv · Jun 13, 2019 · PreprintImmunology