Author

J. E. Clark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Kesson· 1 shared B. J. Cottle· 1 shared C. A. Jones· 1 shared C. C. Blyth· 1 shared C.-X. Li· 1 shared D. Torella· 1 shared E. C. Holmes· 1 shared E. Domenjo-Vila· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMicrobiology

A. Kesson

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Diagnosis and analysis of unexplained cases of childhood encephalitis in Australia using metagenomic next-generation sequencing

C.-X. Li, R. Burrell, R. C. Dale, A. Kesson, C. C. Blyth, J. E. Clark, N. Crawford, C. A. Jones, P. N. Britton, E. C. Holmes

This study uses metagenomic sequencing to identify viral and immune-mediated causes of unexplained childhood encephalitis in Australia, finding respiratory viruses in five cases and immune-mediated causes in six.

bioRxiv · May 10, 2021 · PreprintMicrobiology

Aged-senescent cells contribute to impaired heart regeneration

F. C. Lewis-McDougall, P. J. Ruchaya, E. Domenjo-Vila, T. S. Teoh, L. Prata, B. J. Cottle, J. E. Clark, P. P. Punjabi, W. Awad, D. Torella, T. Tchkonia, J. L. Kirkland, G. M. Ellison-Hughes

This study shows that aged-senescent cardiac progenitor cells impair heart regeneration and that eliminating them improves cardiac function in aged mice.

bioRxiv · Oct 1, 2018Physiology