Author

J. Gondin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Fessard· 1 shared A. Garcia· 1 shared A. Ghasemizadeh· 1 shared A. Guiraud· 1 shared A. Ogier· 1 shared A. Zavoriti· 1 shared B. Chatel· 1 shared B. Chazaud· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 9 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPhysiology

A. Fessard

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cyclosporin A delays the terminal disease stage in Tfam KO mice without improving mitochondrial energy production

B. Chatel, I. Varlet, A. Ogier, E. Pecchi, M. Bernard, J. GONDIN, H. Westerblad, D. Bendahan, C. Gineste

bioRxiv · Oct 14, 2022 · PreprintPathology

Individualized isometric neuromuscular electrical stimulation training promotes myonuclear accretion in mouse skeletal muscle

A. Zavoriti, A. Fessard, M. Rahmati, P. Del Carmine, B. Chazaud, J. GONDIN

This study shows that individualized NMES training induces myonuclear accretion in mouse skeletal muscle without causing damage or hypertrophy.

bioRxiv · Dec 14, 2021 · PreprintPhysiology

Muscle MACF1 maintains myonuclei and mitochondria localization through microtubules to control muscle functionalities

A. Ghasemizadeh, E. Christin, A. Guiraud, N. Couturier, V. Risson, E. Girard, C. Soler, L. Laddada, K. Jagla, C. Sanchez, F.-I. Jaque-Fernandez, A. Garcia, M. Lanfranchi, V. Jacquemond, J. Gondin, , V. Gache

This study identifies MACF1 as a regulator of myonuclei and mitochondria positioning in muscle through microtubules, affecting muscle function and disease.

bioRxiv · May 15, 2019Cell Biology

Annexin A1 drives macrophage skewing towards a resolving phenotype to accelerate the regeneration of muscle injury through AMPK activation

S. McArthur, T. Gobbetti, G. Juban, T. Desgeorges, M. Theret, J. Gondin, J. Toller-Kawahisa, C. Reutelingsperger, M. Perretti, R. Mounier

This study identifies Annexin A1 as a driver of macrophage polarization towards a resolving phenotype via AMPK activation, enhancing muscle regeneration after injury.

bioRxiv · Aug 29, 2018Cell Biology