Author

T. Desgeorges

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

G. Juban· 2 shared J. Zhang· 2 shared K. De Bock· 2 shared M. Theret· 2 shared R. Ardicoglu· 2 shared R. Mounier· 2 shared Z. Fan· 2 shared A. Batavia· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPhysiology

G. Juban

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Single cell compendium of muscle microenvironment in peripheral artery disease reveals altered endothelial diversity and LYVE1+ macrophage activation

G. Turiel, T. Desgeorges, E. Masschelein, Z. Fan, D. Lussi, C. M. Capelle, G. Bernardini, R. Ardicoglu, K. Schönberger, M. Birrer, S. F. Fucentese, J. Zhang, D. Latorre, S. Engelberger, K. De Bock

This study uses single-cell RNA sequencing and spatial transcriptomics to characterize the muscle microenvironment in PAD, identifying altered endothelial diversity and LYVE1+ macrophage activation.

bioRxiv · Jun 21, 2023Cell Biology

The vascular gene Apold1 is dispensable for normal development but controls angiogenesis under pathological conditions

Z. Fan, R. Ardicoglu, A. Batavia, R. Rust, L. von Ziegler, R. Waag, J. Zhang, T. Desgeorges, O. Sturman, H. Dang, R. Weber, A. E. Moor, M. E. Schwab, P.-L. Germain, J. Bohacek, K. De Bock

This study shows that Apold1 is not essential for normal angiogenesis but is critical for revascularization under ischemic conditions and tumor growth.

bioRxiv · Dec 2, 2022 · PreprintCell Biology

AMPKα1 is essential for Glucocorticoid Receptor triggered anti-inflammatory macrophage activation

G. Caratti, T. Desgeorges, G. Juban, M. Koenen, B. Kozak, M. Theret, B. Chazaud, J. P. Tuckermann, R. Mounier

This study shows that AMPKα1 is required for glucocorticoid receptor-mediated anti-inflammatory macrophage activation, linking metabolism to inflammation resolution.

bioRxiv · Jan 2, 2020 · PreprintPhysiology

Annexin A1 drives macrophage skewing towards a resolving phenotype to accelerate the regeneration of muscle injury through AMPK activation

S. McArthur, T. Gobbetti, G. Juban, T. Desgeorges, M. Theret, J. Gondin, J. Toller-Kawahisa, C. Reutelingsperger, M. Perretti, R. Mounier

This study identifies Annexin A1 as a driver of macrophage polarization towards a resolving phenotype via AMPK activation, enhancing muscle regeneration after injury.

bioRxiv · Aug 29, 2018Cell Biology