Author

J. M. Reitsma

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. Moradian· 1 shared D. Collins-McMillen· 1 shared F. Goodrum· 1 shared H. H. Yu· 1 shared J. A. Nelson· 1 shared J. Buehler· 1 shared K. Zarrella· 1 shared M. Hancock· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBiochemistry

A. Moradian

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Author profile →

2 papers

Recent·Most cited

Human Cytomegalovirus UL138 Interaction with USP1 Activates STAT1 in infection

K. Zarrella, P. Longmire, S. Zeltzer, D. Collins-McMillen, M. Hancock, J. Buehler, J. M. Reitsma, S. S. Terhune, J. A. Nelson, F. Goodrum

This study identifies the UL138-UAF1-USP1 interaction in HCMV that activates STAT1 to regulate latency establishment through innate immune signaling.

bioRxiv · Feb 7, 2023Microbiology

Single subunit degradation of WIZ, a lenalidomide- and pomalidomide dependent substrate of E3 ubiquitin ligase CRL4CRBN

H. H. Yu, J. M. Reitsma, M. J. Sweredoski, A. Moradian, S. Hess, R. J. Deshaies

This study identifies WIZ as a novel substrate of the CRL4CRBN E3 ubiquitin ligase complex that is degraded by lenalidomide and pomalidomide, highlighting the mechanism of IMiD-induced protein degradation.

bioRxiv · Apr 2, 2019 · PreprintBiochemistry