Author

J. R. Woodgett

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1

Frequent Co-Authors

B. W. Doble· 1 shared E. M. Jones· 1 shared J. L. Gorman· 1 shared J. R. Adams· 1 shared M. J. Parsons· 1 shared P. S. Ohashi· 1 shared S. E. Egan· 1 shared S. Patel· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 12 mutual co-author links

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1 shared paperImmunology

B. W. Doble

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJul 24, 2019

Regulation of thymocyte β-selection, development and positive selection by glycogen synthase kinase-3

M. J. Parsons, S. Patel, B. W. Doble…·J. R. Woodgett

This study shows that inactivating GSK-3 and GSK-3β in thymocytes disrupts β-selection and positive selection, leading to T cell developmental defects and lymphomas.

DOI 10.1101/712521

mouseflow cytometry

𝕏 PostBluesky

Cancer BiologybioRxivPreprintMar 26, 2019

Development of a drug repressible β-catenin mutant mouse to examine effect of activated β-catenin signaling on long-term maintenance of mammary tumor growth

J. L. Gorman, J. R. Adams, E. M. Jones…·J. R. Woodgett

This study develops a drug-repressible β-catenin mutant mouse model to investigate its role in long-term mammary tumor growth after initiation.

DOI 10.1101/588459

mousegenetic perturbation / CRISPR

𝕏 PostBluesky