Author

J. Russell Moore

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Adam W. ClemensiD· 2 shared Harrison W. GabeliD· 2 shared Alaina UrmaniD· 1 shared Cheryl A. HilliD· 1 shared David F. WozniakiD· 1 shared Dennis Y. WuiD· 1 shared Diana C. BeardiD· 1 shared Diana L. Christian· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Adam W. ClemensORCID

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2 papers

Recent·Most cited

Non-CG DNA methylation and MeCP2 stabilize repeated tuning of long genes that distinguish closely related neuron types

J. Russell Moore, Mati T. Nemera, Rinaldo D. D’Souza, Nicole Hamagami, Adam W. Clemens, Diana C. Beard, Alaina Urman, Victoria Rodriguez Mendoza, Harrison W. Gabel

The paper shows that non-CG DNA methylation and MeCP2 regulate long genes to maintain neuron type-specific gene programs at high resolution.

bioRxiv · Jan 30, 2024 · PreprintNeuroscience

DNMT3A haploinsufficiency results in behavioral deficits and global epigenomic dysregulation shared across neurodevelopmental disorders

Diana L. Christian, Dennis Y. Wu, Jenna R. Martin, J. Russell Moore, Yiran R. Liu, Adam W. Clemens, Sabin A. Nettles, Nicole M. Kirkland, Thomas Papouin, Cheryl A. Hill, David F. Wozniak, Joseph D. Dougherty, Harrison W. Gabel

This study shows that DNMT3A mutations cause behavioral and epigenomic changes in mice, similar to human neurodevelopmental disorders, by disrupting non-CG DNA methylation.

Cell Reports · Nov 1, 2020Peer-reviewedNeuroscience