Author

J. Tarrago-Celada

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

E. Zodda· 2 shared M. Cascante· 2 shared T. M. Thomson· 2 shared A. Suarez-Bonnet· 1 shared C. Foguet· 1 shared C. Hernandez-Carro· 1 shared I. H. Polat· 1 shared J. J. Lozano· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

E. Zodda

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Glutaminase as a metabolic target of choice to counter acquired resistance to Palbociclib by colorectal cancer cells

M. Tarrado-Castellarnau, C. Foguet, J. Tarrago-Celada, M. Palobart, C. Hernandez-Carro, J. Perarnau, E. Zodda, I. H. Polat, S. Marin, A. Suarez-Bonnet, J. J. Lozano, M. Yuneva, T. M. Thomson, M. Cascante

This study shows that combining Palbociclib with Telaglenestat prevents acquired resistance in colorectal cancer cells by counteracting metabolic reprogramming.

bioRxiv · Jan 4, 2024Cancer Biology

Cell-autonomous metabolic reprogramming and oxidative stress underlie endothelial dysfunction in acute myocardial infarction

E. Zodda, O. Tura-Ceide, N. L. Mills, J. Tarrago-Celada, M. Carini, T. M. Thomson, M. Cascante

This study identifies metabolic reprogramming and oxidative stress in endothelial cells from acute myocardial infarction patients as drivers of endothelial dysfunction.

bioRxiv · Feb 28, 2023Cell Biology