Author

T. M. Thomson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:metabolism & bioenergetics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

M. Cascante· 3 shared C. Foguet· 2 shared E. Zodda· 2 shared J. J. Lozano· 2 shared J. Tarrago-Celada· 2 shared S. Marin· 2 shared A. Calvet· 1 shared A. de Lucas-Rius· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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3 shared papersCancer Biology

M. Cascante

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Glutaminase as a metabolic target of choice to counter acquired resistance to Palbociclib by colorectal cancer cells

M. Tarrado-Castellarnau, C. Foguet, J. Tarrago-Celada, M. Palobart, C. Hernandez-Carro, J. Perarnau, E. Zodda, I. H. Polat, S. Marin, A. Suarez-Bonnet, J. J. Lozano, M. Yuneva, T. M. Thomson, M. Cascante

This study shows that combining Palbociclib with Telaglenestat prevents acquired resistance in colorectal cancer cells by counteracting metabolic reprogramming.

bioRxiv · Jan 4, 2024Cancer Biology

Metabolic and mitochondria alterations induced by SARS-CoV-2 accessory proteins ORF3a, ORF9b, ORF9c and ORF10

B. D. Lopez-Ayllon, S. Marin, M. Farinas Fernandez, T. Garcia-Garcia, R. Fernandez-Rodriguez, A. de Lucas-Rius, N. Redondo, L. Mendoza-Garcia, C. Foguet, J. Grigas, A. Calvet, J. M. Villalba, M. J. Rodriguez Gomez, D. Megias, B. Mandracchia, , M. Montoya

This study identifies mitochondrial and metabolic reprogramming induced by SARS-CoV-2 accessory proteins in lung cells, revealing potential therapeutic targets.

bioRxiv · Sep 26, 2023Immunology

Cell-autonomous metabolic reprogramming and oxidative stress underlie endothelial dysfunction in acute myocardial infarction

E. Zodda, O. Tura-Ceide, N. L. Mills, J. Tarrago-Celada, M. Carini, T. M. Thomson, M. Cascante

This study identifies metabolic reprogramming and oxidative stress in endothelial cells from acute myocardial infarction patients as drivers of endothelial dysfunction.

bioRxiv · Feb 28, 2023Cell Biology