Author

J. Van den Eynden

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Research Focus:differential expression· 1 Organism:mouse· 1

Frequent Co-Authors

B. Hallberg· 2 shared A. Aniszewska· 1 shared A. Claeys· 1 shared A. R. Clausen· 1 shared A. Schmidt· 1 shared A. Stahlberg· 1 shared A. Strömberg· 1 shared D. E. Lind· 1 shared

Activity by Year

2017-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCancer Biology

B. Hallberg

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4 papers

Recent·Most cited

Cancer BiologybioRxivSep 17, 2023

Preclinical exploration of the DNA Damage Response pathway using the interactive neuroblastoma cell line explorer CLEAN.

J. Gabre, P. Merseburger, A. Claeys…·J. Van den Eynden

The paper introduces CLEAN, a web tool for exploring neuroblastoma data, focusing on DNA damage response to identify drug targets.

DOI 10.1093/narcan/zcad062

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintAug 30, 2023

ALK signalling primes the DNA damage response sensitizing ALK-driven neuroblastoma to ATR inhibition

M. Borenäs, G. Umapathy, D. E. Lind…·R. H. Palmer

This study identifies ALK signaling's role in enhancing DNA damage response in neuroblastoma, showing that combined ATR/ALK inhibition leads to sustained tumor response and differentiation.

DOI 10.1101/2023.08.30.555570

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivAug 31, 2018

Elevated pyrimidine dimer formation at distinct genomic bases underlie promoter mutation hotspots in UV-exposed cancers

K. Elliott, M. Bostrom, S. Filges…·E. Larsson

The paper shows that UV-induced mutations in cancer promoters are due to base-specific increases in pyrimidine dimer formation, not inhibited repair.

DOI 10.1371/journal.pgen.1007849

humanwhole-genome sequencing

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Cancer BiologybioRxivDec 15, 2017

An antisense RNA capable of modulating the expression of the tumour suppressor microRNA-34a

J. T. Serviss, F. C. Richter, J. Van den Eynden…·K. Pokrovskaja Tamm

This study identifies a long non-coding RNA, lncTAM34a, that modulates miR34a expression and its tumor suppressive functions in human cancers.

DOI 10.1038/s41419-018-0777-5

humanexperimental assay

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