Author

J.-S. Hulot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Bioengineering· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:proteomics· 2

Frequent Co-Authors

A. Sayed· 2 shared C. Jouve· 2 shared G. Marazzi· 2 shared M. Valente· 2 shared A. Achab-Ali· 1 shared A. Georges· 1 shared B. Hoareau· 1 shared C. Cerveau· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 11 mutual co-author links

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2 shared papersCell Biology

A. Sayed

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

A versatile high-throughput assay based on 3D ring-shaped cardiac tissues generated from human induced pluripotent stem cell derived cardiomyocytes

M. Seguret, P. Davidson, S. Robben, C. Jouve, C. Pereira, Q. Lelong, L. Deshayes, C. Cerveau, M. Le Berre, R. S. Rodrigues Ribeiro, J.-S. Hulot

The paper develops a high-throughput 3D cardiac tissue assay using human iPSC-derived cardiomyocytes and fibroblasts to study mechanical and drug responses.

bioRxiv · Mar 26, 2023Bioengineering

Inhibition of integrin alpha V (CD51) reduces inflammation and transition to heart failure following pressure overload

C. Delacroix, A. Achab-Ali, P. Alayrac, M. Gandon-Renard, F. Drame, D. A. Sassoon, J.-S. Silvestre, J.-S. Hulot

This study shows that inhibiting integrin alpha V reduces inflammation and prevents heart failure progression in pressure-overloaded mice by modulating pro-inflammatory macrophages.

bioRxiv · Oct 10, 2022 · PreprintPharmacology & Toxicology

Genomic, transcriptomic and proteomic depiction of iPSC-derived smooth muscle cells as emerging cellular models for arterial diseases

L. Liu, C. Jouve, J. Henry, T.-E. Berrandou, J.-S. Hulot, A. Georges, N. Bouatia-Naji

This study compares two iPSC-derived smooth muscle cell models, showing differences in proliferation, gene expression, and chromatin variants linked to arterial diseases.

bioRxiv · May 1, 2022 · PreprintCell Biology

Hypoxia promotes a perinatal-like progenitor state in the adult murine epicardium

A. Sayed, S. Turoczi, F. Soares-da-Silva, G. Marazzi, J.-S. Hulot, D. D. A. Sassoon, M. Valente

This study shows that hypoxia reactivates progenitor-like properties in adult murine epicardial cells, promoting endothelial fate and multilineage differentiation potential.

bioRxiv · Sep 16, 2021Cell Biology

Fibroadipogenic Progenitors contribute to microvascular repair during skeletal muscle regeneration

D. Ollitrault, V. Buffa, R. Correra, A. Sayed, B. Hoareau, S. Pohle-Kronawitter, S. Stricker, J.-S. Hulot, M. Valente, G. Marazzi, D. Sassoon

This study shows that fibroadipogenic progenitors (FAPs) differentiate into endothelial cells during skeletal muscle regeneration in response to hypoxia and glucose deprivation.

bioRxiv · Sep 6, 2021 · PreprintCell Biology