Author

James Kaminski

University of California, BerkeleyPrimary ORCID profile (0000-0002-5137-7516 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

James KaminskiiD1 paper · Broad Institute

Biology Disciplines

Click to filter author papers

Developmental Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Nir YosefiD· 2 shared Aaron StreetsiD· 1 shared Alexandra SchnelliD· 1 shared Allon WagneriD· 1 shared Ashley R. HooveriD· 1 shared Aviv RegeviD· 1 shared Brandon Schellhaass· 1 shared Chao WangiD· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersGenomics

Nir YosefORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

In Silico Modeling of Metabolic State in Single Th17 Cells Reveals Novel Regulators of Inflammation and Autoimmunity

Allon Wagner, Chao Wang, Johannes Fessler, David DeTomaso, Julián Ávila-Pacheco, James Kaminski, Sarah Zaghouani, Elena Christian, Pratiksha I. Thakore, Brandon Schellhaass, Elliot H. Akama‐Garren, Kerry A. Pierce, Vasundhara Singh, Noga Ron‐Harel, Vivian Paraskevi Douglas, , Nir Yosef

The paper introduces Compass, an algorithm that uses single-cell RNA-Seq and flux balance analysis to characterize metabolic states in Th17 cells, revealing a metabolic switch and novel anti-inflammatory regulators.

Cell · Jul 2, 2021Peer-reviewedGenomics

T cell self-reactivity during thymic development dictates the timing of positive selection

Lydia Kay Lutes, Zoë Steier, Laura Lynn McIntyre, Shraddha Pandey, James Kaminski, Ashley R. Hoover, Silvia Ariotti, Aaron Streets, Nir Yosef, Ellen A. Robey

This study shows that low self-reactive thymocytes have shorter TCR signals, delayed positive selection, and retain preselection gene expression signatures, suggesting ion channels modulate TCR sensitivity during development.

eLife · Apr 22, 2021Peer-reviewedDevelopmental Biology