Author

Jemima Helen

University Hospital Heidelberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Assay:proteomics· 1 Organism:rat· 1 Approach:statistical model· 1

Frequent Co-Authors

Ferdinand AlthammeriD· 3 shared Matthew K. KirchneriD· 2 shared Tim SchubertiD· 2 shared Yuval Podpecan· 2 shared Alexandre CharletiD· 1 shared Aliyu Jabir Muhammad· 1 shared Angel BaudoniD· 1 shared Annabel C. Kleinwaechter· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersNeuroscience

Ferdinand AlthammerORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Magel2 deficiency drives microglial synaptic engulfment and alters neuron-microglia interactions in Prader-Willi syndrome

Jemima Helen, Moritz Claudius Wimmer, Berkan Kanmaz, Tim Schubert, Ingrid Camila Possa‐Paranhos, Felipe Corrêa‐da‐Silva, Yuval Podpecan, Aliyu Jabir Muhammad, Eva M. Eisemann, Nadia Illma, Derya S. Arslan, Megan Iminitoff, Franziska Schommer, Henning Fröhlich, Felix Franke, , Ferdinand Althammer

This study shows that Magel2 deficiency in a mouse model of Prader-Willi syndrome causes microglial structural changes and increased synaptic engulfment, altering neuron-microglia interactions and hypothalamic circuit function.

bioRxiv · Sep 30, 2026 · PreprintNeuroscience

Angiotensin-II drives changes in microglia-vascular interactions in rats with heart failure

Ferdinand Althammer, Ranjan K. Roy, Matthew K. Kirchner, Yuval Podpecan, Jemima Helen, Shaina McGrath, Elba Campos‐Lira, Javier Eduardo Stern

This study shows that Angiotensin-II increases microglia-vascular interactions in rat heart failure models, leading to neuroinflammation that can be mitigated by blocking AngII receptors.

Communications Biology · Nov 19, 2024Peer-reviewedNeuroscience

Oxytocin Gαi signaling-induced amygdala astrocytes processes retraction shapes behavioral stress response

Angel Baudon, Valentin Grelot, Ferdinand Althammer, Kai‐Yi Wang, Clémence Denis, Syed Azmal Ali, Yudong Yan, Fernando Castillo Díaz, Francesca Piacentini, Etienne Clauss Creusot, Volodya Hovhannisyan, Tim Schubert, Annabel C. Kleinwaechter, Jemima Helen, Tom Lakomy, , Alexandre Charlet

The paper shows that oxytocin-induced astrocyte process retraction in the amygdala modulates freezing behavior during stress by enhancing neuronal sensitivity to potassium.

bioRxiv · Oct 7, 2024 · PreprintNeuroscience