Ranjan K. Roy

The paper shows hypovolemia triggers conserved inverse neurovascular coupling in the SON, similar to salt challenge, but this response is not reversed in heart failure like osmotic stimuli.

The paper identifies an asymmetric blood flow pathway between the nucleus of the solitary tract and area postrema in rats, revealing structural and functional differences in their capillary networks.

This study shows that microglial purinergic signaling reverses salt-induced neurovascular polarity in the hypothalamus during heart failure, restoring oxygen levels through adenosine A2A receptor-mediated vasodilation.

This study shows that Angiotensin-II increases microglia-vascular interactions in rat heart failure models, leading to neuroinflammation that can be mitigated by blocking AngII receptors.

This study investigates changes in the oxytocinergic circuit in the central amygdala of rats with chronic heart failure, finding impaired OT release and receptor function.

The paper identifies a new vascular portal pathway from the SCN to the OVLT, showing circadian-regulated blood flow and AVP transport, suggesting a mechanism for circadian signaling.

The paper investigates changes in the PVN-to-CA2 oxytocin pathway in heart failure rats, finding altered oxytocinergic innervation and reduced OTR-expressing astrocytes.

This study investigates how angiotensin II modulates the A-type K+ current in PVN neurons via the MAPK-ERK pathway in rats with heart failure.

This study shows that oxytocin acts on astrocytes in the central amygdala to promote a positive emotional state, challenging the notion that oxytocin only affects neurons.