Author

Jonathan Langdown

Cambridge University Hospitals NHS Foundation Trust

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Amy D. GelinasiD· 1 shared Amy M. MasoniD· 1 shared CHARGE Hemostasis Working Group· 1 shared Charlotte SummersiD· 1 shared Daniel J. SchneideriD· 1 shared David StaceyiD· 1 shared Dirk S. PauliD· 1 shared Edwin R. ChilversiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Amy D. GelinasORCID

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1 paper

Recent·Most cited

Elucidating mechanisms of genetic cross-disease associations: an integrative approach implicates protein C as a causal pathway in arterial and venous diseases

David Stacey, Lingyan Chen, Joanna M. M. Howson, Amy M. Mason, Stephen Burgess, Stephen G. MacDonald, Jonathan Langdown, Harriet McKinney, Kate Downes, Neda Farahi, James E. Peters, Saonli Basu, James S. Pankow, Nathan Pankratz, Weihong Tang, , Dirk S. Paul

The paper identifies a genetic variant in PROCR that influences protein C and factor VII levels, linking it to both reduced coronary artery disease risk and increased venous thromboembolism risk through opposing inflammatory and thrombotic mechanisms.

medRxiv · Mar 16, 2020 · PreprintGenetic & Genomic Medicine