Author

Harriet McKinney

National Health Service

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Approach:benchmark study· 1 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:database / resource· 1

Frequent Co-Authors

Kate DownesiD· 2 shared Adrian Park· 1 shared Albert KoulmaniD· 1 shared Alessandra CabassiiD· 1 shared Amy D. GelinasiD· 1 shared Amy J. FraryiD· 1 shared Amy M. MasoniD· 1 shared Bekir ErgüneriD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersGenomics

Kate DownesORCID

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2 papers

Recent·Most cited

Transcriptional, epigenetic and metabolic signatures in cardiometabolic syndrome defined by extreme phenotypes.

Denis Seyres, Alessandra Cabassi, John J. Lambourne, Frances Burden, Samantha Farrow, Harriet McKinney, Joana Batista, Carly Kempster, Maik Pietzner, Oliver C. Slingsby, Thong Huy Cao, Paulene A. Quinn, Luca Stefanucci, Matthew C. Sims, Karola Rehnström, , Mattia Frontini

The paper identifies molecular and metabolic signatures of cardiometabolic syndrome using extreme phenotype groups and shows changes after bariatric surgery.

Clinical Epigenetics · Mar 12, 2022Peer-reviewedGenomics

Elucidating mechanisms of genetic cross-disease associations: an integrative approach implicates protein C as a causal pathway in arterial and venous diseases

David Stacey, Lingyan Chen, Joanna M. M. Howson, Amy M. Mason, Stephen Burgess, Stephen G. MacDonald, Jonathan Langdown, Harriet McKinney, Kate Downes, Neda Farahi, James E. Peters, Saonli Basu, James S. Pankow, Nathan Pankratz, Weihong Tang, , Dirk S. Paul

The paper identifies a genetic variant in PROCR that influences protein C and factor VII levels, linking it to both reduced coronary artery disease risk and increased venous thromboembolism risk through opposing inflammatory and thrombotic mechanisms.

medRxiv · Mar 16, 2020 · PreprintGenetic & Genomic Medicine