Author

Daniel J. Schneider

SomaLogic (United States)ORCID 0000-0001-6830-0905Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Nebojša JanjićiD· 2 shared Amy D. GelinasiD· 1 shared Amy M. MasoniD· 1 shared CHARGE Hemostasis Working Group· 1 shared Charlotte SummersiD· 1 shared Daniel Raimund BergeriD· 1 shared David StaceyiD· 1 shared Dirk S. PauliD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersNeuroscience

Nebojša JanjićORCID

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2 papers

Recent·Most cited

Probing Molecular Diversity and Ultrastructure of Brain Cells with Fluorescent Aptamers

Xiaotang Lu, Yuelong Wu, Peter H. Li, Tao Fang, Richard Lee Schalek, Yaxin Su, Daniel Raimund Berger, Jeffery D. Carter, Sean Lynch, Daniel J. Schneider, Shashi Gupta, Viren Jain, Nebojša Janjić, Jeff William Lichtman

This paper introduces CAptVE, a method for fluorescent aptamer-based labeling that enables molecular analysis in ultrastructurally reconstructable electron micrographs.

Nature Communications · Apr 27, 2026Peer-reviewedNeuroscience

Elucidating mechanisms of genetic cross-disease associations: an integrative approach implicates protein C as a causal pathway in arterial and venous diseases

David Stacey, Lingyan Chen, Joanna M. M. Howson, Amy M. Mason, Stephen Burgess, Stephen G. MacDonald, Jonathan Langdown, Harriet McKinney, Kate Downes, Neda Farahi, James E. Peters, Saonli Basu, James S. Pankow, Nathan Pankratz, Weihong Tang, , Dirk S. Paul

The paper identifies a genetic variant in PROCR that influences protein C and factor VII levels, linking it to both reduced coronary artery disease risk and increased venous thromboembolism risk through opposing inflammatory and thrombotic mechanisms.

medRxiv · Mar 16, 2020 · PreprintGenetic & Genomic Medicine