Author

K. Bridges

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

K. Miller-Jensen· 2 shared A. Hefner· 1 shared A. Sheth· 1 shared A. Vidyarthi· 1 shared B. Beadle· 1 shared C. R. Pickering· 1 shared D. Molkentine· 1 shared F. M. Johnson· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersImmunology

K. Miller-Jensen

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Functionally and metabolically divergent melanoma-associated macrophages originate from common bone-marrow precursors

G. A. Pizzurro, K. Bridges, X. Jiang, A. Vidyarthi, K. Miller-Jensen, O. R. Colegio

This study identifies functionally and metabolically distinct melanoma-associated macrophages originating from common bone-marrow precursors, with implications for targeted therapy.

bioRxiv · Jun 2, 2023Immunology

Apoptosis recognition receptors regulate skin tissue repair in mice

O. Justynski, K. Bridges, W. Krause, M. F. Forni, Q. Phan, T. Sandoval-Schaefer, R. Driskell, K. Miller-Jensen, V. Horsley

The paper investigates the role of apoptosis recognition receptors in skin wound repair using mouse models and single-cell RNA sequencing, finding that Axl signaling is critical for repair while Timd4 is essential for efferocytosis.

bioRxiv · Jan 17, 2023 · PreprintCell Biology

Inhibition of histone acetyltranserase function radiosensitizes CREBBP/EP300 mutants via repression of homologous recombination, potentially targeting a novel gain of function.

M. Kumar, D. Molkentine, J. Molkentine, K. Bridges, T. Xie, L. Yang, A. Hefner, M. Gao, M. J. Frederick, S. Seth, M. Abdelhakiem, B. Beadle, F. M. Johnson, J. Wang, L. Shen, , H. D. Skinner

The paper identifies CREBBP/EP300 as radiosensitization targets in mutant tumors by inhibiting histone acetyltransferase, which represses homologous recombination and increases radiation sensitivity.

bioRxiv · Apr 10, 2020 · PreprintCancer Biology