Author

K. C. MacNamara

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

G. Fredman· 3 shared C. Decker· 2 shared J. L. Bonin· 2 shared M. Marinello· 2 shared M. Spite· 2 shared S. Sadhu· 2 shared A. Adam· 1 shared A. Hebe Mena· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersImmunology

G. Fredman

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4 papers

Recent·Most cited

Resolvin E1 improves efferocytosis and rescues severe aplastic anemia in mice

R. Grazda, A. N. Seyfried, K. R. Maddipatti, G. Fredman, K. C. MacNamara

This study shows that Resolvin E1 improves efferocytosis and rescues severe aplastic anemia in mice by resolving inflammation and enhancing cell clearance.

bioRxiv · Feb 15, 2023Immunology

The Resolvin D2-GPR18 Axis Enhances Bone Marrow Function and Limits Hepatic Fibrosis in Aging

H. Fitzgerald, J. L. Bonin, S. Sadhu, M. Lipscomb, N. Biswas, C. Decker, M. Nabage, R. Bossardi, M. Marinello, A. Hebe Mena, K. Gilliard, M. Spite, A. Adam, K. C. MacNamara, G. Fredman

This study shows that RvD2-GPR18 signaling reduces hepatic fibrosis and steatosis in aging by enhancing bone marrow function and macrophage populations.

bioRxiv · Jan 5, 2023 · PreprintPhysiology

Co-infection of Ehrlichia with B. burgdorferi drives emergency myelopoiesis and promotes Lyme arthritis

J. L. Bonin, S. R. Torres, A. L. Marcinkiewicz, X. Yang, U. Pal, J. DiSpirito, T. Nowak, Y.-P. Lin, K. C. MacNamara

This study shows that co-infection with Ehrlichia and Borrelia burgdorferi in mice causes arthritis and emergency myelopoiesis, highlighting immune signals over pathogen burden in disease development.

bioRxiv · Aug 23, 2022 · PreprintImmunology

Sub-lethal radiation-induced senescence impairs resolution programs and drives cardiovascular inflammation

S. Sadhu, C. Decker, B. E. Sansbury, M. Marinello, A. Seyfried, J. Howard, M. Mori, Z. Hosseini, T. Arunachalam, A. V. Finn, J. M. Lamar, D. Jourd'heuil, L. Guo, K. C. MacNamara, M. Spite, G. Fredman

This study shows sub-lethal radiation induces macrophage senescence, impairing efferocytosis and promoting cardiovascular inflammation, with RvD1 treatment reversing these effects.

bioRxiv · May 19, 2021Immunology