Author

K. Jacquet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:drug response / drug discovery· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. B. Kimenyi Ishimwe· 1 shared A. Corpet· 1 shared A. Fradet-Turcotte· 1 shared A. Hubstenberger· 1 shared B. Mari· 1 shared B. Mograbi· 1 shared B. Romeo· 1 shared C. Lacoux· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiochemistry

A. B. Kimenyi Ishimwe

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Author profile →

2 papers

Recent·Most cited

The TUDOR domain of SMN is an H3K79me1 histone mark reader

O. Binda, A. B. Kimenyi Ishimwe, M. Galloy, K. Jacquet, A. Corpet, A. Fradet-Turcotte, J. Cote, P. Lomonte

This study identifies SMN's TUDOR domain as an H3K79me1 histone mark reader and shows SMA-linked mutations disrupt this interaction.

bioRxiv · Oct 6, 2022Biochemistry

Processing body dynamics drive non-genetic MEK inhibitors tolerance by fine-tuning KRAS and NRAS translation

O. Vidal-Cruchez, V. J. Nicolini, T. Rete, R. Rezzonico, C. Lacoux, J. Fassy, K. Jacquet, M. A. Domdom, C. Ventujol, T. Juhel, B. Romeo, J. Roux, A. Hubstenberger, B. Mari, B. Mograbi, , P. Brest

The paper identifies a mechanism where MEK inhibitors induce tolerance by promoting KRAS and NRAS translation via PB dissolution, revealing a dynamic feedback loop in MAPK signaling.

bioRxiv · Dec 6, 2021 · PreprintCancer Biology