Author

A. Fradet-Turcotte

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. B. Kimenyi Ishimwe· 1 shared A. Blondeau· 1 shared A. Corpet· 1 shared A. Gravel· 1 shared A. Jablonski· 1 shared C. Vaziri· 1 shared D. Jayaprakash· 1 shared E. Biquand· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperBiochemistry

A. B. Kimenyi Ishimwe

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3 papers

Recent·Most cited

The TUDOR domain of SMN is an H3K79me1 histone mark reader

O. Binda, A. B. Kimenyi Ishimwe, M. Galloy, K. Jacquet, A. Corpet, A. Fradet-Turcotte, J. Cote, P. Lomonte

This study identifies SMN's TUDOR domain as an H3K79me1 histone mark reader and shows SMA-linked mutations disrupt this interaction.

bioRxiv · Oct 6, 2022Biochemistry

The immediate early protein 1 of human herpesvirus 6B counteracts ATM activation in an NBS1-dependent manner

V. Collin, E. Biquand, V. Tremblay, E. G. Lavoie, J. Dessapt, A. Blondeau, A. Gravel, L. Flamand, A. Fradet-Turcotte

This paper shows that HHV-6B IE1 inhibits ATM activation at double-strand breaks via NBS1 interaction, leading to genomic instability and affecting viral replication.

bioRxiv · Jul 31, 2021 · PreprintMicrobiology

A Degenerate PCNA-Interacting Peptide (DPIP) box targets RNF168 to replicating DNA to limit 53BP1 signaling

Y. Yang, D. Jayaprakash, R. Hollingworth, S. Chen, A. Jablonski, E. Mutter-Rottmayer, Y. Gao, J. Anand Ramlal, J. An, X. Chen, K. Pearce, S.-A. Blanchet, A. Fradet-Turcotte, G. Stewart, C. Vaziri

This study identifies a non-canonical role of RNF168 at DNA replication forks, showing it suppresses 53BP1 signaling through PCNA binding, independent of its canonical DSB response function.

bioRxiv · Mar 17, 2021 · PreprintCancer Biology