Author

K. R. Chaudhary

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:rat· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

D. J. Stewart· 2 shared Y. Deng· 2 shared A. J. Duval· 1 shared A. Yang· 1 shared C. G. Broustas· 1 shared C.-J. Lee· 1 shared D. W. Courtman· 1 shared H. B. Lieberman· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersPhysiology

D. J. Stewart

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Author profile →

3 papers

Recent·Most cited

Vicious cycle of hemodynamic perturbation and endothelial injury in development and progression of pulmonary arterial hypertension

Y. Deng, K. R. Chaudhary, A. Yang, K. Kesavan, L. Wang, K. Chathely, D. J. Stewart

This study shows that hemodynamic perturbations and endothelial injury form a cycle driving pulmonary arterial hypertension, which can be reversed by unilateral lung banding.

bioRxiv · Oct 16, 2023 · PreprintPhysiology

Single-cell microencapsulation improves lung retention of endothelial colony forming cells after intravascular delivery and unmasks therapeutic benefit in severe pulmonary arterial hypertension

N. D. Cober, K. R. Chaudhary, Y. Deng, C.-J. Lee, K. Rowe, D. W. Courtman, D. J. Stewart

This study shows that microencapsulation of endothelial colony forming cells (ECFCs) improves their lung retention and therapeutic efficacy in a rat model of pulmonary arterial hypertension.

bioRxiv · Nov 5, 2022Bioengineering

Targeting MEK5 impairs non-homologous end-joining repair and sensitizes prostate cancer to DNA damaging agents

C. G. Broustas, A. J. Duval, K. R. Chaudhary, R. A. Friedman, R. K. Virk, H. B. Lieberman

This study shows that targeting MEK5 impairs NHEJ repair and enhances sensitivity of prostate cancer to DNA-damaging agents in vitro and in vivo.

bioRxiv · Aug 15, 2019Cancer Biology