Author

K. Sugiyama

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Approach:statistical model· 1

Frequent Co-Authors

G. Matyas· 1 shared H. Yanagisawa· 1 shared J. D. Smith· 1 shared J. Meienberg· 1 shared K. J. Kovacs· 1 shared K. Takahashi· 1 shared K. Yoshioka· 1 shared L. Lamberti· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPathology

G. Matyas

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2 papers

Recent·Most cited

CtBP restricts DUX-dependent and -independent genetic program for the 2-cell-like state in murine embryonic stem cells

K. Yoshioka, K. Sugiyama, M. Ichisakino, S. A. Abadi, N. Hayakawa, M. Marutani, R. Masuda, K. Takahashi, Y. Seki

The paper shows that Ctbp1/2 disruption activates DUX-dependent and -independent pathways in mESCs, leading to 2-cell-like state features through PRAMEL7 upregulation.

bioRxiv · Jan 12, 2024Cell Biology

Novel Insights into the Aortic Mechanical Properties of Mice Modeling Hereditary Aortic Diseases

N. Dubacher, K. Sugiyama, J. D. Smith, V. Nussbaumer, M. Csonka, S. Ferenczi, K. J. Kovacs, S. M. Caspar, L. Lamberti, J. Meienberg, H. Yanagisawa, M. B. Sheppard, G. Matyas

This paper measures aortic rupture force in mouse models of hereditary aortic diseases to assess biomechanical integrity and drug effects.

bioRxiv · Aug 15, 2023Pathology