Author

L. D. Cruz-Zaragoza

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:fluorescence / live-cell microscopy· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

P. Rehling· 2 shared A. D. Davidson· 1 shared A. E. Firth· 1 shared A. Krempler· 1 shared A. Valpadashi· 1 shared D. A. Matthews· 1 shared E. Emmott· 1 shared E. Lavdovskaia· 1 shared

Activity by Year

2022-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersMicrobiology

P. Rehling

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologybioRxivNov 15, 2022

The SARS-CoV-2 protein ORF3c is a mitochondrial modulator of innate immunity

H. Stewart, Y. Lu, S. O'Keefe…·A. E. Firth

This study identifies ORF3c as a SARS-CoV-2 protein that localizes to mitochondria and inhibits IFN-β production without affecting NF-κB or JAK-STAT signaling.

DOI 10.1016/j.isci.2023.108080

humanproteomics

𝕏 PostBluesky

BiochemistrybioRxivOct 10, 2022

A quantitative fluorescence-based approach to study mitochondrial protein import

N. Jain, R. Gomkale, O. Bernhard…·L. D. Cruz-Zaragoza

This paper introduces a fluorescence-based method for analyzing mitochondrial protein import with high sensitivity and quantification capabilities.

DOI 10.15252/embr.202255760

fluorescence / live-cell microscopyexperimental assay

𝕏 PostBluesky

BiochemistrybioRxivJul 8, 2022

Human mtRF1 terminates COX1 translation and its ablation induces mitochondrial ribosome-associated quality control

F. Nadler, E. Lavdovskaia, A. Krempler…·R. Richter-Dennerlein

The paper identifies mtRF1 as the specialized release factor for COX1 translation termination in human mitochondria and shows its ablation causes COX deficiency and activates quality control mechanisms.

DOI 10.1038/s41467-022-34088-w

humanproteomics

𝕏 PostBluesky