Author

L. Katsnelson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

D. Fenyo· 2 shared P. Cheng· 2 shared T. Davoli· 2 shared X. Zhao· 2 shared A. F. Johnson· 1 shared A. Goldberg· 1 shared A. Mermerian· 1 shared A. S. Freire· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCell Biology

D. Fenyo

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Author profile →

3 papers

Recent·Most cited

KaryoCreate: a new CRISPR-based technology to generate chromosome-specific aneuploidy by targeting human centromeres

N. Bosco, A. Goldberg, A. F. Johnson, X. Zhao, J. C. Mays, P. Cheng, J. J. Bianchi, C. Toscani, L. Katsnelson, D. Annuar, S. Mei, R. E. Faitelson, I. Y. Pesselev, K. S. Mohamed, A. Mermerian, , T. Davoli

KaryoCreate generates chromosome-specific aneuploidy by targeting centromeres with CRISPR, showing it can induce gains or losses of targeted chromosomes in human cells.

bioRxiv · Sep 27, 2022 · PreprintCell Biology

Proteogenomic analysis of aneuploidy reveals divergent types of gene expression regulation across cellular pathways

T. Davoli, P. Cheng, X. Zhao, L. Katsnelson, R. Moya, J. Shwetar, D. Fenyo

This study uses proteogenomic analysis to show that gene expression regulation differs across cellular pathways and tissues, with protein-level regulation dominating in some pathways and RNA-level regulation in others.

bioRxiv · Dec 7, 2021 · PreprintCell Biology

Endothelial mTOR maintains hematopoiesis during aging

P. Ramalingam, M. G. Poulos, M. C. Gutkin, L. Katsnelson, A. S. Freire, E. Lazzari, J. M. Butler

This study shows that mTOR signaling in bone marrow endothelial cells is critical for maintaining hematopoiesis during aging, and its inhibition accelerates aging-related hematopoietic decline.

bioRxiv · Mar 13, 2020Cell Biology