Author

Leanne N. Ho

University of California, Irvine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:CRISPR screen· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Jennifer T. StocksdaleiD· 2 shared Leslie Michels ThompsoniD· 2 shared Nicolette R. McClureiD· 2 shared Ricardo MiramontesiD· 2 shared Alice Lau· 1 shared Amber Keith· 1 shared Carlos Chillón-Marinas· 1 shared Cholsoon JangiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Jennifer T. StocksdaleORCID

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2 papers

Recent·Most cited

iPSC Neurodegenerative Disease Initiative isogenic CAG repeat iPSC line for Huntingtons disease

Lisa Salazar, Mara S. Burns, Jennifer T. Stocksdale, Keona Q. Wang, Gloria Cao, Ricardo Miramontes, Nicolette R. McClure, Leanne N. Ho, Amber Keith, Margaret L. Sutherland, Mark Cookson, Michael Emmerson Ward, William C. Skarnes, Leslie Michels Thompson

The paper generates isogenic iPSC lines with CAG repeats and HTT KO for Huntington's disease study, showing differentiation potential and altered gene expression in neurons and astrocytes.

Journal of Huntington s Disease · Oct 16, 2026Peer-reviewedNeuroscience

Aberrant splicing in Huntington's disease via disrupted TDP-43 activity accompanied by altered m6A RNA modification.

Thai B. Nguyen, Ricardo Miramontes, Carlos Chillón-Marinas, Roy Maimon, Sonia Vazquez‐Sanchez, Alice Lau, Nicolette R. McClure, Whitney England, Monika K. Singha, Jennifer T. Stocksdale, Ki-Hong Jang, Sunhee Jung, Jharrayne I. McKnight, Leanne N. Ho, Richard L. M. Faull, , Leslie Michels Thompson

This study identifies TDP-43 dysfunction and reduced m6A modification as mechanisms causing aberrant splicing in Huntington's disease using mouse and human brain data.

bioRxiv · Oct 31, 2023 · PreprintNeuroscience