Author

Mara S. Burns

University of California, Irvine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:CRISPR screen· 1 Organism:Drosophila· 1

Frequent Co-Authors

Keona Q. WangiD· 2 shared Leslie Michels ThompsoniD· 2 shared Nicolette R. McClureiD· 2 shared Ricardo MiramontesiD· 2 shared Alice Lau· 1 shared Amber Keith· 1 shared Andrea M. Reyes-OrtiziD· 1 shared Corey J. Schulz· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Keona Q. WangORCID

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2 papers

Recent·Most cited

iPSC Neurodegenerative Disease Initiative isogenic CAG repeat iPSC line for Huntingtons disease

Lisa Salazar, Mara S. Burns, Jennifer T. Stocksdale, Keona Q. Wang, Gloria Cao, Ricardo Miramontes, Nicolette R. McClure, Leanne N. Ho, Amber Keith, Margaret L. Sutherland, Mark Cookson, Michael Emmerson Ward, William C. Skarnes, Leslie Michels Thompson

The paper generates isogenic iPSC lines with CAG repeats and HTT KO for Huntington's disease study, showing differentiation potential and altered gene expression in neurons and astrocytes.

Journal of Huntington s Disease · Oct 16, 2026Peer-reviewedNeuroscience

Integrated transcriptome analysis of Huntingtons disease iPSC-derived and mouse astrocytes implicates dysregulated synaptogenesis, actin, and astrocyte maturation

Andrea M. Reyes-Ortiz, Edsel M. Abud, Mara S. Burns, Jie Wu, Sarah J. Hernandez, Nicolette R. Geller, Keona Q. Wang, Corey J. Schulz, Ricardo Miramontes, Alice Lau, Neethu Michael, Emily Miyoshi, Mathew M. Blurton-Jones, David Van Vactor, John C. Reidling, , Leslie Michels Thompson

This study compares HD astrocytes from human iPSCs and mouse models, identifying dysregulated synaptogenesis, actin signaling, and maturation, with unique human findings and potential therapeutic targets.

bioRxiv · Jul 28, 2022 · PreprintNeuroscience