Author

M. A. Marie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

E. J. Sanderlin· 2 shared L. V. Yang· 2 shared A. Hoffman· 1 shared E. A. Krewson· 1 shared H. Hong· 1 shared J. Velcicky· 1 shared K. D. Cashwell· 1 shared P. Loetscher· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

E. J. Sanderlin

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2 papers

Recent·Most cited

GPR4 knockout attenuates intestinal inflammation and forestalls the development of colitis-associated colorectal cancer in murine models

M. A. Marie, E. J. Sanderlin, A. Hoffman, K. D. Cashwell, S. Satturwar, H. Hong, Y. Sun, L. V. Yang

This study shows GPR4 knockout reduces intestinal inflammation and tumor development in colitis-associated colorectal cancer mouse models.

bioRxiv · Mar 13, 2023 · PreprintCancer Biology

The proton-sensing GPR4 receptor regulates paracellular gap formation and permeability of vascular endothelial cells

E. A. Krewson, E. J. Sanderlin, M. A. Marie, J. Velcicky, P. Loetscher, L. V. Yang

This study shows that GPR4 activation in endothelial cells under acidic conditions increases paracellular gap formation and permeability, mediated by G12/13 signaling, and suggests GPR4 as a therapeutic target for inflammation and edema.

bioRxiv · Apr 6, 2019Cell Biology