Author

M. d. C. Sasiain

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Research Focus:cell signaling & trafficking· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Organism:mouse· 1 Organism:non-human primate· 1

Frequent Co-Authors

C. Verollet· 3 shared L. Balboa· 3 shared O. Neyrolles· 3 shared D. Palmero· 2 shared F. Fuentes· 2 shared G. Lugo-Villarino· 2 shared J. L. Marin Franco· 2 shared M. Genoula· 2 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersImmunology

C. Verollet

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivApr 3, 2023

Elevated glycolytic metabolism of monocytes limits the generation of HIF-1α-driven migratory dendritic cells in tuberculosis

M. Maio, J. Barros, M. Joly…·L. Balboa

This study shows that elevated glycolytic metabolism in monocytes during tuberculosis impairs their differentiation into migratory dendritic cells, limiting immune response.

DOI 10.7554/elife.89319

humanproteomics

𝕏 PostBluesky

Cell BiologyeLifePeer-reviewedMar 30, 2020

Tuberculosis-associated IFN-I induces Siglec-1 on tunneling nanotubes and favors HIV-1 spread in macrophages

C. Verollet, M. Dupont, S. Souriant…·G. Lugo-Villarino

This study identifies Siglec-1 as a factor induced by TB-associated IFN-I that localizes on TNTs and promotes HIV-1 spread in macrophages.

DOI 10.7554/elife.52535

non-human primatebulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivMar 23, 2020

Host-derived lipids from tuberculous pleurisy impair macrophage microbicidal-associated metabolic activity

J. L. Marin Franco, M. Genoula, D. Corral…·L. Balboa

This study shows that host-derived lipids from tuberculous pleurisy reduce glycolytic activity and impair HIF-1-mediated metabolic reprogramming in macrophages, promoting Mtb infection.

DOI 10.1016/j.celrep.2020.108547

mouseproteomics

𝕏 PostBluesky