Author

M. D. Meyer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Lim· 1 shared C. Gopal· 1 shared C. L. Pennington· 1 shared D. Kang· 1 shared E. B. Goff· 1 shared G. V. Echeverria· 1 shared J. Lee· 1 shared J. P. Barrish· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

B. Lim

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Author profile →

3 papers

Recent·Most cited

Cytotoxic rhamnolipid micelles drive acute virulence in Pseudomonas aeruginosa

Q. Xu, D. Kang, M. D. Meyer, C. L. Pennington, C. Gopal, J. W. Schertzer, N. Kirienko

This study shows rhamnolipid micelles from Pseudomonas aeruginosa are highly cytotoxic to host and bacterial cells, correlating with virulence in clinical isolates.

bioRxiv · Oct 13, 2023 · PreprintMicrobiology

The short chain fatty acid propionic acid activates the Rcs stress response system partially through inhibition of D-alanine racemase

N. S. Harshaw, M. D. Meyer, N. A. Stella, K. M. Lehner, R. P. Kowalski, R. M. Shanks

The study identifies propionic acid as an activator of the Rcs stress response in Serratia marcescens, showing it inhibits D-alanine racemase rather than affecting intracellular pH.

bioRxiv · Aug 17, 2022Microbiology

Mitochondrial structure and function adaptation in residual triple negative breast cancer cells surviving chemotherapy treatment

M. L. Baek, J. Lee, K. E. Pendleton, M. J. Berner, E. B. Goff, L. Tan, S. A. Martinez, T. Wang, M. D. Meyer, B. Lim, J. P. Barrish, W. Porter, P. L. Lorenzi, G. V. Echeverria

This study identifies how residual triple negative breast cancer cells adapt mitochondrial structure and function to survive chemotherapy, showing that DNA-damaging agents promote mitochondrial fusion and OXPHOS, which can be targeted with OPA1 inhibitors.

bioRxiv · Feb 25, 2022Cancer Biology