Author

M. M. Mangala

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. P. Hill· 2 shared J. I. Vandenberg· 2 shared C. Semsarian· 1 shared G. Ranpura· 1 shared H. C. Szappanos· 1 shared J. A. Gillis· 1 shared L. C. Hool· 1 shared M. D. Perry· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

A. P. Hill

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Slow conduction and spatial dispersion of repolarization are intrinsic properties of cardiomyocyte electrophysiology that contribute to proarrhythmia in an iPSC model of hypertrophic cardiomyopathy

S. Lim, M. M. Mangala, M. Holliday, H. C. Szappanos, S. Barratt-Ross, W. Liang, G. Ranpura, J. I. Vandenberg, C. Semsarian, A. P. Hill, L. C. Hool

This study shows that HCM cardiomyocytes have slower conduction and increased repolarization dispersion, which contribute to arrhythmia in an iPSC model.

bioRxiv · Jul 20, 2023 · PreprintPhysiology

Co-expression of calcium channels and delayed rectifier potassium channels protects the heart from proarrhythmic events

S. Ballouz, M. M. Mangala, M. D. Perry, S. Heitmann, J. A. Gillis, A. P. Hill, J. I. Vandenberg

The paper identifies co-expression of CACNA1C and KCNH2 in cardiac cells as protective against proarrhythmic events through reduced action potential variability.

bioRxiv · Mar 11, 2020 · PreprintBiophysics