Author

M. M. S. van Lohuizen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. R. R. Maia· 1 shared G. Chandrasekaran· 1 shared G. Karakulah· 1 shared G. Ozgun· 1 shared N. Landman· 1 shared S. Erkek Ozhan· 1 shared S. Gisler· 1 shared S. Senturk· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. R. R. Maia

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintApr 19, 2023

Retinoids and EZH2 inhibitors cooperate to orchestrate cytotoxic effects on bladder cancer cells

G. Ozgun, T. Yaras, N. Landman…·S. Erkek Ozhan

This study shows that combining retinoids and EZH2 inhibitors enhances cytotoxic effects on bladder cancer cells by promoting apoptosis, cell cycle arrest, and anti-oncogenic gene expression.

DOI 10.1101/2023.04.19.537500

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivDec 24, 2019

A genome-wide enrichment screen identifies NUMA1-loss as a resistance mechanism against mitotic cell-death induced by BMI1 inhibition

M. M. S. van Lohuizen, S. Gisler, A. R. R. Maia, G. Chandrasekaran

The paper identifies NUMA1 loss as a resistance mechanism against BMI1 inhibitor-induced mitotic cell death through a genome-wide screen in human cells.

DOI 10.1371/journal.pone.0227592

humangenetic perturbation / CRISPR

𝕏 PostBluesky