Author

M. P. Jedrychowski

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Easton· 1 shared A. Gikandi· 1 shared A. J. Aguirre· 1 shared A. M. Whiteley· 1 shared A. S. Chandhoke· 1 shared B. Małachowska· 1 shared D. A. Scott· 1 shared D. Finley· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

A. Easton

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Author profile →

2 papers

Recent·Most cited

Global proteomics of Ubqln2-based murine models of ALS

A. M. Whiteley, M. A. Prado, S. A. H. de Poot, J. A. Paulo, M. Ashton, S. Dominguez, M. Weber, H. Ngu, J. Szpyt, M. P. Jedrychowski, A. Easton, S. P. Gygi, T. Kurz, M. J. Monteiro, E. J. Brown, D. Finley

The paper identifies proteomic changes in Ubqln2 mutant mice and finds that TRIM32, PEG10, and CXX1B are linked to UBQLN2 function and degradation.

bioRxiv · Feb 22, 2020Cell Biology

Defining and targeting adaptations to oncogenic KRASG12C inhibition using quantitative temporal proteomics

N. Santana-Codina, A. S. Chandhoke, Q. Yu, B. Małachowska, M. Kuljanin, A. Gikandi, M. Stanczak, S. Gableske, M. P. Jedrychowski, D. A. Scott, A. J. Aguirre, W. Fendler, N. S. Gray, J. D. Mancias

This study uses quantitative temporal proteomics to analyze KRASG12C inhibitor responses in cancer models, identifying effective drug combinations to overcome resistance.

bioRxiv · Sep 14, 2019Cancer Biology