Author

M. S. Sim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:bacteria (other)· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Mulay· 1 shared A. Purkayastha· 1 shared A. S. Bradley· 1 shared B. Konda· 1 shared B. N. Gomperts· 1 shared B. R. Stripp· 1 shared C. Sen· 1 shared D. W. Shia· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Mulay

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Energy flux couples sulfur isotope fractionation to proteomic and metabolite profiles in Desulfovibrio vulgaris

W. D. Leavitt, J. Waldbauer, S. S. Venceslau, M. S. Sim, L. Zhang, F. J. Boidi, S. Plummer, J. M. Diaz, I. A. C. Pereira, A. S. Bradley

The paper links sulfur isotope fractionation to proteomic and metabolite profiles in Desulfovibrio vulgaris under different energy regimes, showing how energy flux affects protein abundance and isotopic composition.

bioRxiv · Aug 27, 2023 · PreprintMicrobiology

Direct exposure to SARS-CoV-2 and cigarette smoke increases infection severity and alters the stemcell-derived airway repair response

A. Purkayastha, C. Sen, G. Garcia, J. Langerman, P. Vijayaraj, D. W. Shia, L. K. Meneses, T. M. Rickabaugh, A. Mulay, B. Konda, M. S. Sim, B. R. Stripp, K. Plath, V. Arumugaswami, B. N. Gomperts

This study shows that cigarette smoke exposure increases SARS-CoV-2 infection severity in airway cultures by reducing interferon response and ABSC proliferation.

bioRxiv · Jul 28, 2020Cell Biology