Author

M. W. Khan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:metabolomics· 1

Frequent Co-Authors

B. T. Layden· 2 shared B. Wicksteed· 2 shared M. Priyadarshini· 2 shared A. Sharma· 1 shared A. Terry· 1 shared B. P. Bernabe· 1 shared B. Szynal· 1 shared B.-A. Mittermuller· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

B. T. Layden

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Role of socioeconomic factors and interkingdom crosstalk in the dental plaque microbiome in early childhood caries

M. W. Khan, V. C. de Jesus, B.-A. Mittermuller, S. Sareen, V. Lee, R. J. Schroth, P. Hu, P. Chelikani

This study examines the dental plaque microbiome in early childhood caries, finding associations between specific microbes, socioeconomic factors, and host variables.

bioRxiv · Mar 12, 2024Microbiology

Discovering Disease Genes in PPI Networks: A Bridge from Centrality to Communities

M. W. Khan, R. A. Shams Malick, H. Cherifi

This paper introduces a method to identify key genes in disease networks by analyzing community-aware centrality, finding novel genes in prostate and cervical cancer.

bioRxiv · Sep 8, 2023 · PreprintCancer Biology

The hexokinase ''HKDC1'' interaction with the mitochondria is essential for hepatocellular carcinoma progression

M. W. Khan, A. Terry, M. Priyadarshini, G. Guzman, J. Cordoba-Chacon, I. Ben-Sahra, B. Wicksteed, B. T. Layden

This study shows that HKDC1's interaction with mitochondria is critical for HCC progression by altering metabolism and causing mitochondrial dysfunction.

bioRxiv · Nov 10, 2021 · PreprintCancer Biology

Gestational insulin resistance is mediated by the gut microbiome-indoleamine 2,3-dioxygenase axis

M. Priyadarshini, G. Navarro, D. Reiman, A. Sharma, K. Xu, K. Lednovich, C. R. Manzella, M. W. Khan, B. Wicksteed, G. E. Chlipala, B. Szynal, B. P. Bernabe, P. M. Maki, R. K. Gill, J. Gilbert, , B. T. Layden

This study identifies the gut microbiome-indoleamine 2,3-dioxygenase (IDO1) axis as a mediator of gestational insulin resistance in mice.

bioRxiv · Jul 21, 2021Physiology