Author

M.-B. Madel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Blin-Wakkach· 3 shared A. Wakkach· 2 shared D. MOULIN· 2 shared H.-J. Garchon· 2 shared J. Halper· 2 shared L. Ibanez· 2 shared M. Rouleau· 2 shared M. Topi· 2 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersPhysiology

C. Blin-Wakkach

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Specific targeting of inflammatory osteoclastogenesis by the probiotic yeast S. boulardii CNCM I-745 reduces bone loss in osteoporosis

M.-B. Madel, J. Halper, L. Ibanez, C. Lozano, M. Rouleau, A. Boutin, R. Pontier-Bres, T. Ciucci, M. Topi, C. Hue, J. Amiaud, S. Iborra, D. Sancho, D. Heymann, H.-J. Garchon, , C. Blin-Wakkach

This study identifies PRRs regulating inflammatory osteoclasts and shows that S. boulardii reduces bone loss by targeting these cells.

bioRxiv · Aug 1, 2022Physiology

Osteoclasts contribute to early development of chronic inflammation by promoting dysregulated hematopoiesis and myeloid skewing

M.-B. Madel, L. Ibanez, T. Ciucci, J. Halper, M. Topi, H.-J. Garchon, M. Rouleau, C. G. Mueller, L. Peyrin Biroulet, D. MOULIN, C. Blin-Wakkach, A. Wakkach

This study shows osteoclasts drive myeloid skewing and inflammation in chronic colitis by altering HSC niche interactions and promoting myelopoiesis.

bioRxiv · Dec 9, 2020 · PreprintImmunology

Adipose-tissue derived signals control bone remodelling.

M.-B. Madel, H. Fu, D. Pierroz, M. Schiffrin, C. Winkler, A. Wilson, C. Pochon, B. Toffoli, J.-Y. Jouzeau, F. Gilardi, S. Ferrari, N. Bonnet, C. BLIN-WAKKACH, B. Desvergne, D. MOULIN

This study shows that adipose-derived signals, particularly adiponectin, regulate bone remodeling by inhibiting osteoclastogenesis and that AdipoRon can treat inflammatory bone loss.

bioRxiv · Mar 2, 2020 · PreprintCell Biology