Author

Mark X. Li

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Andrew J. KuehiD· 1 shared Andrew Webb· 1 shared Boris Reljic· 1 shared Catherine Chang· 1 shared Cathrine M. HalliD· 1 shared Colin HockingsiD· 1 shared Daniel H. D. Gray· 1 shared David C. S. Huang· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Andrew J. KuehORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

BAX requires VDAC2 to mediate apoptosis and to limit tumor development

Hui San Chin, Mark F. van Delft, Robert L. Ninnis, Mark X. Li, Iris K. L. Tan, Boris Reljic, Kristen Scicluna, Laura F. Dagley, Jarrod J. Sandow, Gemma L. Kelly, Stephane Chappaz, Seong L. Khaw, Catherine Chang, Andrew Webb, Colin Hockings, , Grant Dewson

This study identifies VDAC2 as essential for BAX-mediated apoptosis and tumor suppression, showing BAX and BAK are differentially regulated.

bioRxiv · Feb 16, 2018 · PreprintCell Biology