Author

Martin Schwaderer

University of Freiburg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 2

Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Ralf GilsbachiD· 2 shared Adam R. Jacobs· 1 shared Björn Andreas GrüningiD· 1 shared Bruce D. GelbiD· 1 shared C Cao· 1 shared Christian Braun· 1 shared David Kranzhöfer· 1 shared Deepak Prabhu RamanujamiD· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersGenomics

Ralf GilsbachORCID

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2 papers

Recent·Most cited

In vivo silencing of regulatory elements using a single AAV-CRISPRi vector

Patrick Laurette, C Cao, Deepak Prabhu Ramanujam, Martin Schwaderer, T. Lueneburg, S Kuss, L. Weiss, Ramile Dilshat, Eileen E. M. Furlong, Flávia Rezende, Stefan Hanns Engelhardt, Ralf Gilsbach

This paper develops an AAV-based CRISPRi system to silence gene expression in cardiac myocytes, showing dose-dependent reduction of Nppa expression in vitro and in vivo.

bioRxiv · Nov 5, 2023 · PreprintGenomics

Distinct epigenetic programs regulate cardiac myocyte development and disease in the human heart in vivo

Ralf Gilsbach, Martin Schwaderer, Sebastian Preißl, Björn Andreas Grüning, David Kranzhöfer, Pedro Schneider, Thomas Georg Nührenberg, Sonia Mulero‐Navarro, Dieter Weichenhan, Christian Braun, Martina Dreßen, Adam R. Jacobs, Harald Lahm, Torsten Doenst, Rolf Backofen, , Lutz Hein

This study examines the dynamics of the cardiac myocyte epigenome during development and in chronic heart failure, revealing distinct epigenetic programs in health and disease.

Nature Communications · Jan 22, 2018Peer-reviewedGenomics